3/10/2023 0 Comments Splenic flexture syndrom![]() Prior stress also enhances the response to subsequent inflammatory stimuli. In animals, stress causes a reactivation of previous enteric inflammation and induces the attendant physiological changes. Stress has also been implicated as a modulator of gastrointestinal inflammation in both animal and human studies. ![]() Some of these observations have been made in human studies of irritable bowel syndrome (IBS) in patients after an enteric infection or in patients in remission from ulcerative colitis. In some instances, these changes persist long after resolution of the mucosal inflammation. Studies in animals have shown that inflammation of the mucosa of the gastrointestinal tract is accompanied by changes in enteric nerve and smooth muscle function, and in gut motility and sensation. The treatment should be based on the principles of ICS treatment. SFS can be considered as a variant of ICS arising in HFSA. Provoking factors of the symptoms are stress situations in childhood. The pain corresponds most to clinical criteria of irritable colon syndrome diagnosis and is accompanied with reduced threshold of pain sensitivity to balloon extension. ![]() Some HFSA patients have pain on the left side-splenic flexure syndrome (SFS). HFSA is encountered mainly in persons with hyperstenic constitution. the results of the following examinations were analysed: the disease history, detailed intestinal investigation, rectal sensitivity, constitution, connective tissue weakness, vegetative dysfunction and regulation. The study group included 82 patients with HFSA, the comparison group-76 patients with irritable colon syndrome (ICS), control group-19 patients without colon pathology. To investigate phenotypical and clinical characteristics in individuals with high fixation of the splenic angle (HFSA). ![]()
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